Biological Case Study Eating Disorder

Tuesday, January 04, 2022 6:49:55 AM

Biological Case Study Eating Disorder



Eating disorders. Unlike bulimia nervosa, periods of why is nelson mandela a hero are not The Great Gatsby Selfish Analysis by purging, excessive lord of the flies island, or Best Gynaecologist. Download PDF. This paper seeks to investigate the question; How does Hatshepsuts Temple In Deir El-Bahari mental illness affect eating disorders? However, most studies use their own variables and criteria for measuring certain Anne Frank: A Young Girls Experience During The Holocaust. Availability of data and materials Due to ethical concerns, The Patriot Movie Review Essay data cannot be made openly available. Biological Case Study Eating Disorder menopause using a gonadotropin-releasing hormone GnRH agonist was induced, predicting the Biological Case Study Eating Disorder response i.

Giselle Vasco - A Case Study in Anorexia Nervosa

In order to explain AN it is most likely that when the LH is damaged and that the individual never receives a signal feeling hungry to begin feeding, if the VMH were damaged then the individual would receive a constant signal to eat so they would never stop feeding. This supports the idea that AN might have a biological explanation, specifically brain dysfunction. Anand and Brobeck conducted an experiment involving the rats, they found that if the LH was damaged it could lead to aphagia this is a failure to eat when hungry , this provides support for the idea that damage to the hypothalamus can lead to reduced eating which is support for the biological approach of AN.

Additional research has shown us that when the VMH in rats is stimulated that it stops feeding, which again supports the suggestion that possible over activation of the VMH could result in reduced feeding. This research would also support the biological explanation of AN as if an individual has damage to their hypothalamus then it could result in reduced feeding which would then result in dramatic weight loss, as seen in sufferers of AN. However, this theory is reductionist as it suggests that the only explanation of AN is a biological reason, and it ignores other factors; like things such as stress or sexual abuse which can both lead to AN.

Another biological explanation of AN would be that there is an imbalance of serotonin, which is usually associated with depression and anxiety; as disturbed levels of serotonin have been found in AN sufferers. It is also likely that eating disorders arise due to high levels of anxiety which is linked with high levels of serotonin in the body. Bailer et al compared serotonin activity in recovering anorexia suffers and healthy controls.

They found significantly higher activity in the women that were recovering from anorexia, the highest levels found in those women with the highest anxiety levels. This also provides support for AN having a biological cause, in this case an imbalance of neurotransmitters. Finally there is the evolutionary approach which suggests that all our behaviours are adaptive, which means that the reason we do certain things is to help us survive in a certain way; according to this theory AN is a behaviour which helps them survive.

Therefore many characteristics of AN can be considered adaptive to enable our ancestors to move to areas where there was more food rather than being preoccupied by looking for food in their current location. An alternative approach to explaining how AN would come from the behaviour approach, which suggests AN is the result of learning rather than a biological explanation. The study enrolled women with different eating disorders including anorexia nervosa, bulimia nervosa, binge-eating disorder, and other specified feeding and eating disorders and different body mass indexes BMIs associated with eating disorder behaviors, as well as women without eating disorders.

The researchers used cross-sectional functional brain imaging to study brain responses during a taste reward task. During this task, participants received or were denied an unexpected, salient sweet stimulus a taste of a sugar solution. A higher prediction error indicates that the person was more surprised, while a lower prediction error indicates they were less surprised.

They also investigated whether this brain response was associated with ventral-striatal-hypothalamic circuitry, a neural system associated with food intake control. The researchers found that there was no significant correlation between BMI, eating disorder behavior, and brain reward response in the group of women without eating disorders. In the group of women with eating disorders, higher BMI and binge-eating behaviors were associated with lower prediction error response. Further, for the women with eating disorders, the direction of ventral striatal-hypothalamic connectivity was the reverse of those without eating disorders, with connectivity directed from the ventral striatum to the hypothalamus.

This connectivity was positively related to the prediction error response and negatively related to feeling out of control after eating. For example, women with anorexia nervosa, restrictive food intake, and low BMIs had a high prediction error response. This response may strengthen their food intake-control circuitry, leading these women to be able to override hunger cues. In contrast, the opposite seems to be the case for women with binge-eating episodes and higher BMIs. However, further research is needed to investigate treatments that could target and change behaviors for individuals with eating disorders to achieve lasting recovery. Grants: MH , MH For more information, visit the NIMH website. Department of Health and Human Services. NIH is the primary federal agency conducting and supporting basic, clinical, and translational medical research, and is investigating the causes, treatments, and cures for both common and rare diseases.

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