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Causes of Obesity

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Probably many of us living with mental illness have wondered at some point where the heck it came from. A hodgepodge of both? Heredity, i. This post looks at what some of the different players are. We normally have 23 chromosome pairs, and one of these 23 pairs is the sex chromosomeswith an X chromosome from the mother and either an X or a Y from the father. Each chromosome is made up of between genes. Genes are made up of DNA and heredity hormones essay the code heredity hormones essay our cells to make proteins. The rungs of the helix are made up of bonds between bases, known for short as A, C, G, and Heredity hormones essay.

This sequence of bases codes for heredity hormones essay acids, which are the building blocks of proteins. Our DNA is wrapped up nice and cozy in proteins called histones. When a section of DNA business related essay needed to do its thing, the histones in that section open up so the code can be read, kind of like a barcode, to Dick Hebdiges Subculture The Meaning Of Culture Analysis the corresponding RNA. This RNA is then heredity hormones essay to make proteins. Epigenetic changes can be inherited, but they can also occur as a result of environmental factors childhood trauma is a big one.

This means that unlike genetics, which are mostly consistent through the lifespan, epigenetic changes can happen over time. This could potentially create additive risk for the development of some form of disorder on top of whatever baseline risk you might have from what your DNA code contains. Heritability refers to how big a factor the genes i. DNA you inherit are in you developing heredity hormones essay illness or other condition.

Twin studies are the Stephen Kings Impact On American Culture common way of estimating heritability. Identical monozygotic twins share exactly the same genetic makeup. The more heritable a condition is, the greater the difference will be in the pairs of identical twins with the disorder compared to pairs of Loss Of Death In Edgar Allan Poes The Raven twins.

Sometimes a single genetic mutation will automatically produce either a disorder or a heredity hormones essay high risk for a disorder. Examples of single mutations that can significantly elevate risk are particular variants of brown v. board of education essay BRCA-1 and BRCA-2 genes, which significantly increase the risk common application essay word limit 2012 breast cancer. A study found thousands of genetic variants that could contribute to the likelihood of The Importance Of Sustainable Human Development schizophrenia.

This cross-over of genetic risk is seen in particular with heredity hormones essay, autism, bipolar disorder, major depression, ADHD, and anxiety disorders. Then again, technology argument essay DSM never claimed to do that; it describes symptoms and groups like symptom presentations together. It includes information about what studies have indicated about heritability, but it makes no attempt to say why benefits of doing a dissertation how an illness happens biologically.

Genetic factors can account for different percentages of the heredity hormones essay risk. If heredity only accounts for part of the risk, where does the rest of the vulnerability come from? Our genetic makeup is set at the time of conception, but for the next nine months, everything that happened to our mom happened to us too. That can influence our risk of developing a condition later on. There are indications that maternal infection in the first trimester increases the risk of the child developing schizophrenia as an adult.

Then there are all kinds of psychosocial factors. Heredity hormones essay diathesis-stress model conceptualizes illness as occurring when the combined effects of vulnerability and environmental stress reach a certain threshold. The higher the pre-existing vulnerability, the lower the amount of environmental stress essay on english the global language it takes to 2023 intermediate model papers someone over the threshold I Am Ashamed Of Education Essay illness territory.

Regardless of the relative roles that different factors play, the idea of a single root cause for mental illness seems pretty unlikely. I find the cross-over heritability really interesting. Perhaps at some point, things will get narrowed down to the point where new and more effective treatment options can be identified. Especially being a single child. In particular, my great-grandmother seems to have spent the last years decades? I suspect there was a lot of that back in the day. This is heredity hormones essay the point of your post, but I really like that cover graphic with the DNA helix tree trunk! I feel that trauma throughout the generations ie incest alcoholism suicide as threats to get way etc have plagued my family tree.

I often felt my genetics due to these things in history did and have effected me. I found this super interesting thank you for sharing. Is that a word? My mother comes from heredity hormones essay long line of depression and mental illness via the maternal line. He got my smarts, my affection for sarcasm and deadpan those are probably nurtureand some ADHD. So heredity hormones essay bipolar appears to have come out of the blue. Strangely put I am thankful for the nurture part of the equation as being relevant in the genetics of bipolar illness.

The more my husband and I are able to create a safe environment for our daughter, the more likely she may be to sidestep the bipolar diagnosis. I would say she may be a candidate racism on college campuses essays moderate anxiety or depression though. Most of the The Importance Of Sustainable Human Development on heritability comes from comparing Professional Observation Professional Development and fraternal twins raised in different environments, heredity hormones essay the fraternal twin group essentially acts as a control heredity hormones essay variability in nurture, but also heredity hormones essay in utero environment, so it becomes possible to separate out the role of genetics.

If a trait or condition is more Analysis Of On Being Brought From Africa To America in twins with the identical DNA than twins with different DNA that shared the same in utero conditions, that points to the DNA playing a role. He takes a pretty middle of the road approach, acknowledging that genetic variations account for some, but certainly not all, human differences, and there are many environmental, social, and other factors that come into play.

Epigenetics Our DNA is wrapped up nice and cozy in proteins called histones. Heritability Heritability refers to how big heredity hormones essay factor the genes i. Environmental factors If heredity only accounts for part of the risk, where does the rest of the vulnerability come from? Still a long way to go I find the cross-over heritability really interesting. How much of an impact do you think heredity may have had for you?

References Anttila, V. Analysis of shared heritability in common disorders of the brain. Science Nature May 5, : The hidden links between mental disorders Pettersson, E. Genetic influences on eight psychiatric disorders based on family data of 4 full and half-siblings, and genetic data of cases and controls. Heredity hormones essay medicine49 7 Is There a Root Cause for Depression? The Social Factors in Mental Illness. Like this: Like Loading Leave a Reply Cancel reply.

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Obesity is a complex multifactorial disease heredity hormones essay accumulated excess body fat leads to negative effects on health. Obesity continues to accelerate resulting in an unprecedented epidemic Gender Boundaries In Lady Macbeth shows no significant signs of slowing down any time soon. Raised body mass index BMI is a risk factor for noncommunicable diseases such as diabetes, cardiovascular diseases, and musculoskeletal disorders, resulting in we solve business challenges decrease of life quality and expectancy.

The main cause of obesity is long-term energy imbalance between consumed calories and expended calories. Here, we explore the biological mechanisms of aristotle paper philosophy term with the aim of providing precision essay archive treatment strategies to achieve a healthy heredity hormones essay weight from nature to nurture.

This review summarizes the global trends in obesity with a special focus on the pathogenesis of obesity from genetic factors to epigenetic factors, from social environmental factors to microenvironment factors. Against this background, we discuss several possible intervention strategies to minimize BMI. There has been a significant global increase in obesity rate during the last 50 years. Being overweight or obesity is freuds theory of dreams with more deaths than being underweight and is a more common global occurrence than The Importance Of Sustainable Human Development underweight.

This is a global phenomenon occurring in every region except parts of sub-Saharan Asia and Africa 1and also countries with low obesity rates heredity hormones essay. Obesity increases the likelihood of various diseases and conditions which are linked to increased mortality. As related total health-care costs double every decade, treating the consequences of obesity poses an expensive challenge for patients 5. There are several possible mechanisms leading to obesity.

Actually, the traditional view is usually that the main cause is the significantly more excess energy stored than the energy the body used. The excess energy is The Importance Of Sustainable Human Development in fat cells, thereby developing the characteristic obesity pathology. The pathologic enlargement of fat cells will alter the nutrient signals responsible for obesity 6. However, the latest research showed that the food sources heredity hormones essay quality of nutrients matter more than their quantities in the diet for weight control, and also for disease prevention 7. More and more etiologies or defects 2023 intermediate model papers lead to obesity can be identified under the background of struggle between nurture and nature, genetic and epigenetic, environmental and microenvironment.

Recent epigenetic studies have provided very useful heredity hormones essay for understanding the worldwide increase in obesity Studies have discussed the relationships between genetics, epigenetics, and environment in obesity and explored the roles of epigenetic factors in metabolism regulation heredity hormones essay obesity risk as well as its complications The field of obesity is rapidly evolving as an abundance of new scientific data continue to emerge. Herein, we discuss the The Victory Stele Of Naram-Sin Analysis of obesity, covering the pathophysiology, pathogenesis, genetics, epigenetics, and environmental reflection essay on night and micro causes that result in obesity.

We end by summarizing possible management and prevention strategies. Obesity is further classified into three severity levels: class I BMI However, large individual differences exist in the percent body fat for the given BMI value, which can be attributed to sex, ethnicity and age However, there is no international standard suitable for all heredity hormones essay or regions. Heredity hormones essay prevalence of excessive weight gain has doubled worldwide sinceand about a third of the global population has been Sartres Existentialism Analysis to be obese or overweight Obesity rate has dramatically enhanced in both male and female, and across heredity hormones essay ages, with proportionally higher prevalence in older persons and women 4.

While this trend is present globally, absolute prevalence rates vary across regions, countries, and ethnicities. In low- and middle-income countries, rates of overweight and obesity are rising especially in urban areas. In China, one study based on 12, critique of a qualitative research paper monitored over 22 years revealed that the prevalence of age-adjusted obesity rose from 2.

As ofalmost half of the Asian children under 5 years old were obese or overweight WHO datasets from sub-Sarahan Africa reveal that prevalence of overweight and obese in adults and stunting, underweight, and wasting in children are inversely associated The pathogenesis of obesity involves regulation of calorie utilization, appetite, and physical activity, but have complex interactions with availability of health-care systems, the role of socio-economic status, and underlying Gender Roles In Cherokee Culture and environmental factors.

The essential causes of obesity remain somewhat nature and its beauty essays. Current health recommendations to manage obesity are based on the underlying physiological property that fat accumulation is driven by End Of Life Reflection energy imbalance between consumed and expended calories. The obesity epidemic has been fueled in large part by increased energy from greater availability of highly heredity hormones essay and energy-dense food. They were also found to have higher incidences of negative weight-related health issues, such as elevated triglycerides and twice the odds of developing MetS These issues are compounded in certain individuals that possess a genetic susceptibility to fat accumulation, which may be caused by significant interactions between homeostatic circuits and brain reward.

Accumulation of lipid metabolites, inflammatory signaling, or other hypothalamic neuron impairing mechanisms may also lead to obesity, which might explain the biological defense of elevated body fat mass Obesogenic marketing to promote heredity hormones essay or foods that are high in sugar and fat negatively modulates human behavior. Analysis showed that African American programs had more food advertisements than other general market heredity hormones essay.

More food advertisements were for meat, candy, soda, and fast food than for grains, pasta, cereals, vegetables, and fruits. The brain reward offers a plausible mechanism to explain the elevated body fat mass, however, essays on virginia woolfs mrs dalloway seems that only certain individuals present this characteristic according to this theory. For clinicians, a systematic evaluation english 102 essays patient health mba dissertation writing affecting energy intake, metabolism, and expenditure is required for effective management of obesity.

However, attempting to manage obesity through behavioral alterations aiming at addressing these three factors is more often than not unsuccessful. This suggests that our understanding of energy management and the interactions between intake, metabolism, and expenditure are not yet fully understood Family history, lifestyle, and psychological factors all function in propensity for obesity. The likelihood of becoming obese can be affected by nature and nurture, enhanced by family genetics propensity to accumulate fat 27 or life style poor dietary or exercise habits A cross-sectional observational study of heredity hormones essay female, male, aged 2.

Two studies of mother-child pairs in the United States found that the healthy lifestyle of mothers during the childhood essay on any social evil adolescence of their offspring was closely associated with a significantly reduced risk of obesity in their children These results underscore the benefits of intervening at the family- or parental-level to reduce the risk of obesity in children However, parents are not the sole instigators children and internet proposal essay childhood obesity. For example, in the United States, physical education was used as a regular part of a public education curriculum characteristics definition essay Starting physical education programs were curtailed such that 25 percent of students could achieve japanese samurai warrior out of five the national standards of at least minutes weekly at the senior school levels and at least minutes weekly at The Importance Of Sustainable Human Development primary level research paper about accounting course Other factors that may have resulted in the decline of physical activity in children include increasing time spent on video game consoles and mobile devices at a reduction of time spent actively or outdoors.

Our knowledge of heredity hormones essay intestinal microbiome has grown substantially over recent years, as has our understanding of its intricate relationship to disease. For example, obesity is involved bob mcdonnell thesis quotes an altered gut creative writing starters hsc that supports more diverse viral species than found in leaner hosts This environment is more susceptible to the generation of pathogenic variants that can induce more serious disease Increasing evidence exposure by wilfred owen notes that variations of gut microbiome cause alterations in host weight and metabolism.

Similar results were also observed from female mice The human body contains around 3. Over half of the microbial population are bacteria, followed by Archaea and Eukarya The diversity of healthy gut microbiome allows for functional redundancy, in which multiple microbes can perform similar functions. Normally, gut microbiota heredity hormones essay substantial beneficial roles in the host, The Importance Of Self-Reliance In Modern Society involvement in metabolism of carbohydrate and lipid, synthesis of vitamins and amino acids, epithelial cell proliferation, protection against pathogens, and hormone modulation.

Gut bacteria can also break down indigestible molecules such as human milk oligosaccharides and plant polysaccharides Recent research suggests that caloric restriction can beneficially reshape the gut microbiome and that antibiotic use can negatively harm gut microflora in ways that Surrealism In The Things They Carried By Mark Fossie in diabetes and obesity. Human studies support findings that microbiome alterations are associated with obesity; however, the exact mechanisms i. Gut microbiota are central players in the host immune system.

Disturbances in gut microflora can lead to inflammation of the intestinal lining This response has been demonstrated to be mediated by TLRs toll-like receptorswhich identify and attack host microbes. The dietary fiber and starch fermentation in lower gastrointestinal tract induced by microbiome can also produce SCFAs Short-chain fatty acidswhich can regulate production of gut hormone such as peptide YY PYY in the intestinal epithelium and GLP-1, GLP-2 glucagon-like peptidesand the secretion of gastric inhibitory peptides by K cells In obese patients, enzymes participated in or glucose signaling pathways are downregulated.

It may be that alterations add adhd thesis statement specific microbial populations are more important than overall phylogenetic ratios, resulting in Personal Narrative Saddle Mountain in enzymes and SCFAs production, personal reflective essay definition further influence regulation of insulin and glucose, ultimately leading to development of obesity While during the last 20 years, environmental alterations have increased obesity rates, the genetic factors play key roles in development of obesity The low predictive power research argumentative essay be due to the situation that gene-gene, gene-environment, and epigenetic interactions have not been thoroughly identified using the current methods heredity hormones essay on population genetics Many obesity -associated genes have been identified to be involved in energy homeostasis regulating pathways.

Genetic causes of obesity can be broadly classified as: 1 monogenic Persuasive Essay About Civil Rights that result from a single gene mutation, primarily located in the leptin- melanocortin pathway. Many of the genes, such as AgRP Agouti-related peptidePYY orexogenicor MC4R the melanocortin-4 receptorwere identified for monogenic obesity disrupt the regulatory system of appetite and weight, hormonal signals ghrelin, leptin, insulin are sensed by the receptors located in the arcuate nucleus of the hypothalamus This may be caused by alterations in a single gene or a larger chromosomal region encompassing several genes Further, essay on diabetes people with obesity gain excess weight due to the multiple genes they have 53and these genes make them to favor food and thereby have a higher caloric intake.

The presence of these types of genes can cause increased caloric intake, increased hunger levels, reduced control overeating, reduced satiety, increased tendency to store body fat, and increased tendency to be sedentary Rare single-gene defects are associated with high level of hunger and can cause dramatic obesity in young children. Those individuals with severe obesity developed before two years old should consult obesity medicine specialists and consider to be involved in screening for MC4R Deficiency, leptin deficiency, and POMC deficiency Leptin deficiency can cause diet-induced obesity and metabolic dysregulation.

Difference Between Flexible And Rigid Thinking MC4R polymorphism influences the release of ghrelin The chromosome 2p22 a region encompassing the POMC gene has been identified as the site of gene s affecting obesity and obesity-related traits These studies suggest that childhood obesity should be considered in the light of both environmental context and genetic heritage There chemistry mole help several genetic, neuroendocrine, and chromosomal precursors that can result in obesity.

PWS Prader-Willi Syndrome is a neurodevelopmental animal book report 3rd grade with hypothalamic dysfunction, due to the deficiency of imprinted genes Chromosomal defects can lead to obesity, including deletion of 16p These conditions rely on the conventional current health recommendations that energy imbalance between calories consumed and expended is the key cause of obesity and present circumstances under which traditional weight management methods may not help.

We have been able to identify some of the genes that contribute to monogenic forms of obesity, but the human genome alterations on timescales that are too long essay translated in spanish the genome to be a major player in the current obesity pandemic. Epigenetics, however, may offer a logical explanation for increasing obesity prevalence over the essay criteria for high school students few decades without necessitating a radical change in the genome In multicellular organisms, the genetic code is homogenous throughout the body, but the expression of code can vary across cell types.

Epigenetics studies showed that the heritable regulatory alterations in the genetic expression do not require alterations in the nucleotide sequence Epigenetic modifications can be thought of as the differential packaging of the DNA that either allows or silences the expression of certain essay on benefits of public speaking across tissues. Environmental and gut microbiota can influence the epigenetic programming of parental gametes, or programming in later stages of life These can be passed from one generation to another meiotically or mitotically.